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To study the effect of varying plasma lipid and apolipoprotein B (apoB) concentrations on apoB secretion, a synthetic proteinase inhibitor was administered intravenously to six patients with familial combined hyperlipidaemia, six patients with heterozygous familial hypercholesterolaemia and six healthy subjects. A bolus of the inhibitor had no effect on total or very low density lipoprotein triglyceride levels in the normal subjects, but caused a decrease in very low density lipoprotein triglyceride levels in the hypertriglyceridaemic patients, whereas it had no effect on triglyceride levels of low density lipoprotein (LDL) or high density lipoprotein (HDL) in any of the subjects. Baseline values of apoB (0.39 +/- 0.19 mg/100 ml of plasma) and triglyceride (2.06 +/- 0.91 g/100 ml) in the hypertriglyceridaemic patients were significantly higher than those of the normolipidaemic subjects (apoB 0.17 +/- 0.08 mg/100 ml, triglycerides 1.23 +/- 0.22 g/100 ml) (P less than 0.001) or the hyperlipidaemic patients (apoB 0.21 +/- 0.08 mg/100 ml, triglycerides 2.30 +/- 0.87 g/100 ml) (P less than 0.001). The administration of the inhibitor did not significantly alter the proportionate increments in plasma apoB or triglyceride during infusion of heparin; however, the amount of increment of apoB from the baseline was significantly lower in the hypertriglyceridaemic patients than that

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